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Hypertensive retinopathy - MedlinePlus Medical Encyclopedia
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Hypertensive retinopathy

Contents of this page:

Illustrations

Hypertensive retinopathy
Hypertensive retinopathy
Retina
Retina

Definition    Return to top

Hypertensive retinopathy is damage to the back part of the eye (retina) caused by high blood pressure.

Causes    Return to top

High blood pressure can cause damage to blood vessels in the eyes. The higher the blood pressure and the longer it has been high, the more severe the damage is likely to be.

Your health care provider can see narrowing of blood vessels, and excess fluid oozing from blood vessels, with an instrument called an ophthalmoscope. The degree of retina damage (retinopathy) is graded on a scale of 1 to 4.

At grade 1, no symptoms may be present. Grade 4 hypertensive retinopathy includes swelling of the optic nerve and of the visual center of the retina (macula). Such swelling can cause decreased vision.

Other retinopathies that are known complications of high blood pressure are called:

Symptoms    Return to top

Exams and Tests    Return to top

Treatment    Return to top

Control of high blood pressure (hypertension) is the only treatment for hypertensive retinopathy.

Outlook (Prognosis)    Return to top

Patients with grade 4 (severe hypertensive retinopathy) frequently also have heart and kidney complications of high blood pressure. The retina will generally recover well if the blood pressure is controlled, but some patients with grade 4 hypertensive retinopathy will have permanent damage to the optic nerve or macula.

Possible Complications    Return to top

When to Contact a Medical Professional    Return to top

Go to the emergency room or call the local emergency number (such as 911) if you have high blood pressure and vision changes or headaches occur.

Prevention    Return to top

Control of high blood pressure prevents changes in the blood vessels of the eye.

References    Return to top

Klig JE. Ophthalmologic complications of systemic disease. Emerg Med Clin North Am. 2008 Feb;26(1):217-31, viii

Update Date: 9/7/2008

Updated by: David C. Dugdale, III, MD, Professor of Medicine, Division of General Medicine, Department of Medicine, University of Washington School of Medicine. Also reviewed by David Zieve, MD, MHA, Medical Director, A.D.A.M., Inc.

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